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| anthocyanin_deletion [2014/09/01 19:15] – chris.bottoms | anthocyanin_deletion [2014/09/02 19:19] (current) – created chris.bottoms |
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| <div class="figure"> | <div class="figure"> |
| <a href="http://images.maizegdb.org/db_images/Variation/mgn/7101_3161_0702_77.jpg"> | <a href="http://images.maizegdb.org/db_images/Variation/mgn/7101_3161_0702_76.jpg"> |
| <img src="http://images.maizegdb.org/db_images/Variation/mgn/480_w_ds/7101_3161_0702_77.jpg"/> | <img src="http://images.maizegdb.org/db_images/Variation/mgn/480_w_ds/7101_3161_0702_76.jpg"/> |
| </a> | </a> |
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| anthocyanin deletion | anthocyanin deletion |
| </span> | </span> |
| a-x1 def/Ring A1-b; Ear from the cross a1-p/a1-x1 X a1-x1/a1-x1/Ring A-b ear showing colorless shrunken sectors indicating loss of ring-carrying A1-b and Sh2. | chromosome segment carrying A1 Sh2 normal albino and lethal alleles whose recessive null phenotypes are expressed at the loss of a covering ring chromosome carrying the respective dominant allele of these loci. Photo of an ear from, the cross of a1-p Sh2/a1-x3 (a viable heterozygote), X a1-x3/a1-x3/Ring A-b (viable homozygous deficiency because it is covered by the A1-b ring that carries the normal alleles of the lost loci) showing colored kernels with colorless shrunken sectors, colored kernels with pale nonshrunken sectors (loss of ring A1-b) with the a1-x3 and and a1-p female contribution. |
| </div> | </div> |
| </div> | </div> |
| <html> | <html> |
| <div class="figure"> | <div class="figure"> |
| <a href="http://images.maizegdb.org/db_images/Variation/mgn/7101_3161_0702_76.jpg"> | <a href="http://images.maizegdb.org/db_images/Variation/mgn/7101_3161_0702_77.jpg"> |
| <img src="http://images.maizegdb.org/db_images/Variation/mgn/480_w_ds/7101_3161_0702_76.jpg"/> | <img src="http://images.maizegdb.org/db_images/Variation/mgn/480_w_ds/7101_3161_0702_77.jpg"/> |
| </a> | </a> |
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| anthocyanin deletion | anthocyanin deletion |
| </span> | </span> |
| chromosome segment carrying A1 Sh2 normal albino and lethal alleles whose recessive null phenotypes are expressed at the loss of a covering ring chromosome carrying the respective dominant allele of these loci. Photo of an ear from, the cross of a1-p Sh2/a1-x3 (a viable heterozygote), X a1-x3/a1-x3/Ring A-b (viable homozygous deficiency because it is covered by the A1-b ring that carries the normal alleles of the lost loci) showing colored kernels with colorless shrunken sectors, colored kernels with pale nonshrunken sectors (loss of ring A1-b) with the a1-x3 and and a1-p female contribution. | a-x1 def/Ring A1-b; Ear from the cross a1-p/a1-x1 X a1-x1/a1-x1/Ring A-b ear showing colorless shrunken sectors indicating loss of ring-carrying A1-b and Sh2. |
| </div> | </div> |
| </div> | </div> |
